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MilliporeSigma

TMS-002

Sigma-Aldrich

EmbryoMax® L-Glutamine Solution (100X), 200mM

The EmbryoMax L-Glutamine Solution (100X), 200mM is available in a 100 mL format and may be used for routine mouse embryonic stem cell culture applications.

Synonym(s):

L-Glutamine solution

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About This Item

UNSPSC Code:
12352205
eCl@ss:
32160801
NACRES:
NA.75
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Quality Level

form

liquid

manufacturer/tradename

Specialty Media
EmbryoMax®

technique(s)

cell culture | stem cell: suitable

input

sample type: mouse embryonic stem cell(s)
sample type induced pluripotent stem cell(s)

General description

EmbryoMax® L-Glutamine Solution (100X), 200mM may be used for routine mouse embryonic stem cell culture applications.

Application

EmbryoMax® L-Glutamine Solution (100X), 200mM has been used as a component of the high glucose Dulbecco′s modified Eagle medium (DMEM) to culture human hepatic stellate cell line and mouse hippocampal neuronal cell line. It has also been used as a supplement in Roswell Park Memorial Institute (RPMI) media for the maintenance of mouse dendritic cells.

Preparation Note

Store at -20°C for 1 year.

Legal Information

EmbryoMax is a registered trademark of Merck KGaA, Darmstadt, Germany

Disclaimer

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

Storage Class

12 - Non Combustible Liquids

wgk_germany

nwg

flash_point_f

Not applicable

flash_point_c

Not applicable


Certificates of Analysis (COA)

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Ernest T Chivero et al.
Frontiers in cell and developmental biology, 8, 573-573 (2020-08-28)
MicroRNA-124 (miR-124), a brain-enriched microRNA, is known to regulate microglial quiescence. Psychostimulants such as cocaine have been shown to activate microglia by downregulating miR-124, leading, in turn, to neuroinflammation. We thus rationalized that restoring the levels of miR-124 could function

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