SAB1305487
MONOCLONAL ANTI-CD138 antibody produced in mouse
clone 480CT5.4.3, crude ascites, buffered aqueous solution
Synonym(s):
CD_antigen=CD138, Flags: Precursor, SDC, SDC1, Syndecan-1
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About This Item
UNSPSC Code:
12352203
NACRES:
NA.41
biological source
mouse
Quality Level
conjugate
unconjugated
antibody form
crude ascites
antibody product type
primary antibodies
clone
480CT5.4.3, monoclonal
form
buffered aqueous solution
mol wt
32462 Da
species reactivity
human
technique(s)
western blot: 1:1000-1:8000
isotype
IgG1
NCBI accession no.
UniProt accession no.
shipped in
wet ice
storage temp.
−20°C
target post-translational modification
unmodified
Gene Information
human ... SDC1(6382)
Physical form
Supplied in PBS with 0.09% (W/V) sodium azide
Disclaimer
Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.
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Storage Class Code
10 - Combustible liquids
Flash Point(F)
Not applicable
Flash Point(C)
Not applicable
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Xiu-Feng Hu et al.
Molecular medicine reports, 10(3), 1409-1415 (2014-07-16)
The present study aimed to determine whether the expression levels of midkine (MK) and syndecan‑1 correlate with the malignant progression and poor prognosis of gastric cardiac adenocarcinoma (GCA). GCA tissue samples (n=72) were obtained from the Department of Pathology of
Laleh Talebian et al.
Transfusion, 54(6), 1515-1521 (2014-01-23)
The NKG2D receptor, one of the natural killer (NK) cell-activating receptors, is expressed on the surface of CD3+CD8+ T cells, γδ+ T cells, NK cells, NKT cells, and a few CD4+ T cells. We show, for the first time, a
Benjamin R Umiker et al.
European journal of immunology, 44(10), 3093-3108 (2014-07-22)
Systemic lupus erythematosus (SLE) is an autoimmune disease characterized by the presence of pathogenic IgG antinuclear antibodies. Pathogenic IgG autoantibody production requires B-cell activation, leading to the production of activation-induced deaminase (AID) and class switching of IgM genes to IgG.
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